Is now at
Argyll Park, Dixon, California


Patients with common type of muscular dystrophy affecting adults.

DM1 hails from complicated genetic mutations that result in abnormalities of the muscle proteins, including the muscle-specific chloride channel ClC-1. Related StoriesApoE4-carrying males with Alzheimer's disease vulnerable to brain bleedsUCSF-led experts map out melanoma's genetic trajectoriesUnderstanding how schizophrenia affects workings of the brainWorking beneath the theory that abnormal inclusion of a region of the ClC-1 gene known as exon 7a in the ClC-1 mRNA may play a role in the advancement of DM1, Charles Thornton and his colleagues at the University of Rochester, Rochester, discovered a robust approach to correcting this aberrant exon 7a inclusion and reversing myotonia in mouse types of DM1.Nixon, president of the Institute for Cancer Avoidance in NY, is quoted as saying Minerals and vitamins are probably more or less worthless so far as cancer prevention can be involved. This is the sort of statement that characterizes the malignancy industry – – one which is normally profoundly ignorant of the human relationships between nutrition and cancers and demonstrates the shocking level of nutritional illiteracy that exists in our nation’s medical professionals. The known fact is that tumor has everything related to vitamins, minerals, foods, and nourishment. In fact, most of the malignancy we are viewing today is caused by cancer-promoting food ingredients such as sodium nitrate, hydrogenated natural oils, MSG, artificial food colors and various chemical preservatives.